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991.
Rony Zachariah Nathalie Guillerm Selma Berger Ajay M. V. Kumar Srinath Satyanarayana Karen Bissell Mary Edginton Sven Gudmund Hinderaker Katie Tayler‐Smith Rafael Van den Bergh Mohammed Khogali Marcel Manzi Anthony J. Reid Andrew Ramsay John C. Reeder Anthony D. Harries 《Tropical medicine & international health : TM & IH》2014,19(9):1068-1075
992.
Daniel R Perez Nancy E Kemeny Karen T Brown Alexandra N Gewirtz Philip B Paty William R Jarnagin Michael I D'Angelica 《HPB : the official journal of the International Hepato Pancreato Biliary Association》2014,16(8):744-748
Background
Hepatic arterial infusion (HAI) chemotherapy is an effective treatment for patients with liver malignancy. Extrahepatic perfusion (EHP) after HAI pump placement requires correction prior to starting chemotherapy. The aim of this study was to define the origin of arterial branches causing EHP in order to determine if alterations in surgical technique during pump placement might prevent EHP.Methods
A prospectively maintained, single-centre HAI database was reviewed for all patients (2008–2011) with EHP. The origin of arterial branches causing EHP was classified anatomically and patient outcomes were analysed.Results
Of the 327 patients with pumps implanted, 24 evidenced EHP. The arterial branch responsible for EHP perfused the duodenum, pancreas and/or stomach. The branch responsible for EHP arose from the proper hepatic artery (PHA), 1st, 2nd, or 3rd order hepatic artery branches in 7, 10, 5 and 2 patients, respectively. The majority of branches beyond the PHA causing EHP (13/17) originated from the right hepatic artery. In 18 patients, aberrant branches were successfully treated with embolization.Conclusion
These findings provide the anatomic basis for prevention of up to one-third of the cases of EHP intra-operatively, decreasing the number of patients who will require additional procedures for correction of EHP post-operatively. 相似文献993.
Karen Sliwa Denise Hilfiker‐Kleiner Alexandre Mebazaa Mark C. Petrie Aldo P. Maggioni Vera Regitz‐Zagrosek Maria Schaufelberger Luigi Tavazzi Dirk J. van Veldhuisen Jolien W. Roos‐Hesslink Ajay J. Shah Petar M. Seferovic Uri Elkayam Karin van Spaendonck‐Zwarts Katrin Bachelier‐Walenta Frederic Mouquet Elisabeth Kraigher‐Krainer Roger Hall Piotr Ponikowski John J. V. McMurray Burkert Pieske 《European journal of heart failure》2014,16(5):583-591
994.
995.
996.
Andrew M. Lippa Karen E. Ocwieja Julie Iglesias Rima Fawaz Scott Elisofon Christine Lee Tanvi S. Sharma 《Transplant infectious disease》2020,22(4)
A 20‐year‐old male presented 3.5 years after intestinal transplantation with rapidly progressive sensorineural hearing loss. Initial brain imaging was consistent with inflammation and/or demyelination. Lumbar puncture was initially non‐diagnostic and a broad infectious workup was unrevealing. Three months after presentation, a repeat LP detected JC virus for which tests had not earlier been conducted. He continued to deteriorate despite withdrawal of prior immunosuppression and addition of mirtazapine, maraviroc, and steroids. He died of progressive neurologic decompensation 5 months after his initial presentation. This case highlights progressive multifocal leukoencephalopathy (PML) as a rare complication after solid organ transplantation and acute sensorineural hearing loss as an unusual first presenting symptom of PML. JC virus should be considered in the differential diagnosis of acute sensorineural hearing loss in any immunocompromised patient. 相似文献
997.
998.
Jorge E Osorio Ivan D Velez Cynthia Thomson Liliana Lopez Alejandra Jimenez Aurelia A Haller Shawn Silengo Jaclyn Scott Karen L Boroughs Janae L Stovall Betty E Luy John Arguello Mark E Beatty Joseph Santangelo Gilad S Gordon Claire Y-H Huang Dan T Stinchcomb 《The Lancet infectious diseases》2014,14(9):830-838
999.
1000.
William F. Pendergraft III Frank B. Cortazar Julia Wenger Andrew P. Murphy Eugene P. Rhee Karen A. Laliberte John L. Niles 《Clinical journal of the American Society of Nephrology》2014,9(4):736-744