尖牙高型咬合板治疗磨牙症的临床研究(附210例报告)

采用尖牙高型咬合板治疗磨牙症２１０例，男１２１例，女８９例，年龄１２～６５岁，平均２７．９岁，疗程１～３年，平均１５个月。结果所有患者的磨牙症状均得到了有效的控制，磨动的幅度和磨损程度逐渐减小，１１６人治愈（５５．２％）、５９人显效（２８．１％）、３５人有效（１６．７％）。讨论了磨牙症的病因和尖牙高型咬合板治疗磨牙症的机理，认为该咬合板设计科学、制作简单、戴用方便、效果明显，有推广应用价值。     

尖牙高菶型咬合板治疗磨牙症的远期疗效观察

采用自行设计的尖牙高型咬合板治疗磨牙症 ,对 6 8例患者进行 6年以上随访观察。结果 30人治愈 (4 4.1% )、2 2人显效 (32 .4% )、16人有效 (2 3.5 % )。成年患者的颌位及咬合关系无改变。认为尖牙高型咬合板是治疗磨牙症的有较好疗效的方法。     

尖牙高He型咬合板治疗磨牙症的临床研究

采用尖牙高Ｈｅ型咬全板治疗睡症２１０例，男１２１例，女８９例，年龄１２－６５岁，平均２７．９岁，疗程１－３年，平均１５个月。结果所有患者的磨牙症状均得到了有效的控制，磨动的幅度和磨损程度逐渐减小，１１６人治愈，５９人显效，３５人有效。     

尖牙高(牙合)型咬合板治疗磨牙症的远期疗效观察

采用自行设计的尖牙高 型咬合板治疗磨牙症,对68例患者进行6年以上随访观察.结果30人治愈(44.1%)、22人显效(32.4%)、16人有效(23.5%).成年患者的颌位及咬合关系无改变.认为尖牙高 型咬合板是治疗磨牙症的有较好疗效的方法.     

白天磨牙症1例报告

白天磨牙症１例报告北京丰台铁路中心医院口腔科周瑞丰１９９５年１０月我科遇到１例白天磨牙症病例，现报告如下。患者姚××，女，７７岁。１９９５年１０月１６日就诊。自述半年前开始，白天上下牙时有不自主叩齿动作，几天后变为磨牙。有意张口时磨牙停止，一合口不久...     

咬合板治疗磨牙症的磨牙信息监测研究

目的　应用自行研制的磨牙症监测仪客观评价稳定型咬合板与尖牙高型咬合板治疗磨牙症的临床效果。方法　将 2 0名磨牙症患者随机分为两组 ,在受试条件基本一致的情况下 ,于低于息止颌位 0 5mm的高度分别制作稳定型咬合板与尖牙高型咬合板。应用自行研制的磨牙症监测仪监测每名患者治疗前后的磨牙时间与磨牙次数。结果　尖牙高型咬合板组患者磨牙时间与磨牙次数明显减少 ,治疗前后差异有极显著性 ;稳定型咬合板组患者治疗前后的监测数据差异无显著性。结论　磨牙症监测仪能自动测量和记录戴咬合板的磨牙症患者的磨牙数据 ,有一定的临床应用价值 ;尖牙高型咬合板治疗磨牙症的临床效果明显优于稳定型咬合板。     

尖牙高He型咬合板治疗磨牙症的远期疗效观察

采用自行设计的尖牙高Ｈｅ型咬合板治疗磨牙症,对６８例患者进行６年以上随访观察。结果３０人治愈（４４．１％）、２２人显效（３２．４％）、１６人有效（２３．５％）。成年患者的颌位及咬合关系无改变。认为尖牙高Ｈｅ型咬合板是治疗磨牙症的有效疗效的方法。     

稳定型咬合板配合咬合重建治疗咀嚼肌疼痛疗效评价

目的　评价稳定型咬合板配合咬合重建治疗咀嚼肌疼痛的疗效。方法　选取2016年9月至2018年5月于中国医科大学附属口腔医院综合科及修复一科行稳定型咬合板及咬合重建序列治疗的咀嚼肌疼痛患者20例。分析治疗前（T0期）、佩戴咬合板3个月后（T1期）、咬合重建完成即刻（T2期）及咬合重建完成3个月后（T3期）的疼痛视觉模拟评分（visual analogue scale,VAS）、肌电图及Friction颞下颌关节紊乱指数的变化。结果　T1、T2、T3期患者VAS评分、下颌姿势位时颞肌咬肌的肌电活性、颞肌咬肌不对称指数及Friction颞下颌关节紊乱指数均较T0期显著降低,差异有统计学意义（均P < 0.05）;最大紧咬牙时颞肌咬肌的肌电活性较T0期明显增高,差异有统计学意义（均P < 0.05）。T1、T2、T3期患者之间的VAS评分、颞肌咬肌的肌电活性、颞肌咬肌不对称指数及Friction颞下颌关节紊乱指数差异无统计学意义（均P > 0.05）。结论　稳定型咬合板可有效治疗咀嚼肌疼痛,咬合重建序列治疗能很好维持其疗效。     

NTI-tss和稳定咬合板治疗磨牙症的多导睡眠监测研究

目的比较NTI-tss咬合板和稳定咬合板（OS）对于磨牙症的短期疗效。方法10例磨牙症患者采用随机自身交叉对照试验,戴用NTI-tss和OS治疗各1周,于戴用咬合板前一晚、戴用当晚和戴用1周后进行多导睡眠监测,计算磨牙指数和睡眠微觉醒指数,数据采用SAS 9.1混合效应模型进行分析。结果两种咬合板在戴用前、戴用当晚、戴用1周后的睡眠微觉醒指数均无明显差异（P>0.05）。两种咬合板戴用后,磨牙指数均降低。NTI-tss戴用前、戴用当晚和1周后的磨牙指数分别为（7.50±1.11）、（3.45±1.22）、（3.51±1.03）次·h-1,戴用当晚与戴用前、戴用1周与戴用前的差异均有统计学意义（t=26.52,t=26.12,P<0.01）。OS戴用前、戴用当晚和1周后的磨牙指数分别为（7.44±1.23）、（2.97±0.91）、（6.43±1.02）次·h-1,戴用当晚与戴用前、戴用1周与戴用前的差异也有统计学意义（t=16.79,t=3.79,P<0.01）,但与NTI-tss相比,降低程度较小。结论NTI-tss和OS对于睡眠微觉醒的发生无影响,两者都可以降低磨牙指数,但前者对于夜磨牙症的短期疗效可能更稳定。     

Fluoxetine-induced sleep bruxism in an adolescent treated with buspirone: a case report

Sleep bruxism, the involuntary grinding and/or clenching of teeth during sleep, may occur in young children and may be secondary to medication use, especially selective serotonin reuptake inhibitors (SSRIs). The mesocortical disinhibition produced by SSRIs may lead to dopamine depletion that manifests itself as nocturnal bruxism, a specific form of akathisia. This may be prevented by using buspirone, a 5-HT1A agonist that reduces serotonergic activity and increases dopaminergic activity. This article reports on a case history of an adolescent with fluoxetine-induced bruxism that was successfully treated with buspirone. As SSRI use is on the rise for treating childhood psychiatric disorders, this case report highlights the importance of recognizing SSRI-induced bruxism and the possible related adverse dental side effects. Furthermore, this report supports the efficacy of a treatment strategy in adolescents, which has previously been reported only for adult patients.     

Electromyogram biofeedback training for daytime clenching and its effect on sleep bruxism

Bruxism contributes to the development of temporomandibular disorders as well as causes dental problems. Although it is an important issue in clinical dentistry, no treatment approaches have been proven effective. This study aimed to use electromyogram (EMG) biofeedback (BF) training to improve awake bruxism (AB) and examine its effect on sleep bruxism (SB). Twelve male participants (mean age, 26·8 ± 2·5 years) with subjective symptoms of AB or a diagnosis of SB were randomly divided into BF (n = 7) and control (CO, n = 5) groups to undergo 5‐h daytime and night‐time EMG measurements for three consecutive weeks. EMG electrodes were placed over the temporalis muscle on the habitual masticatory side. Those in the BF group underwent BF training to remind them of the occurrence of undesirable clenching activity when excessive EMG activity of certain burst duration was generated in week 2. Then, EMGs were recorded at week 3 as the post‐BF test. Those in the CO group underwent EMG measurement without any EMG BF training throughout the study period. Although the number of tonic EMG events did not show statistically significant differences among weeks 1–3 in the CO group, events in weeks 2 and 3 decreased significantly compared with those in week 1, both daytime and night‐time, in the BF group (P < 0·05, Scheffé's test). This study results suggest that EMG BF to improve AB tonic EMG events can also provide an effective approach to regulate SB tonic EMG events.     

Management of a child with autism and severe bruxism: a case report

Autism is a developmental disorder characterized by severe deficits in social interaction and communication. A wide spectrum of medical and behavioral symptoms is exhibited by children with autism, which makes routine dental care very difficult in them. Bruxism or forceful grinding of teeth is one of the sleep problems commonly observed in children with autism. Our patient, a 4-year-old male child with autism, presented with complaints of pain and sensitivity of the teeth. There was history of excessive grinding and clenching of teeth. Limited oral examination revealed severe attrition of all primary teeth. Treatment was planned under general anesthesia because of his poor cognitive abilities. Full-mouth rehabilitation, including placement of stainless steel crowns for all primary molars, was done. Following treatment there was a significant decrease in the grinding habit over the next 2 months. Although the communication and behavioral problems in children with autism pose challenges for the dentist, treatment with proper planning and a lot of patience can definitely make a difference.     

Masseter muscle nodule due to bruxism. A case in point

A 16-year-old white boy had a soft subcutaneous mass in the right masseter muscle. The pediatrician and two surgeons the patient had previously consulted had disagreed as to the origin of the mass and its treatment; their recommendations had ranged from leaving it alone to surgical excision. Treatment involved an orthopedic oral appliance restoring vertical dimension to its genetic norm and galvanic muscle stimulation. After 1 year the mass was eliminated.     

A vibratory stimulation-based inhibition system for nocturnal bruxism: a clinical report

For the single subject tested to date, the bruxism-contingent vibratory-feedback system for occlusal appliances effectively inhibited bruxism without inducing substantial sleep disturbance. Whether the reduction in bruxism would continue if the device no longer provided feedback and whether the force levels applied are optimal to induce suppression remain to be determined.